Case Study, 3 pages (550 words)

Patho case study

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What type of reaction Is the health care provider concerned about and why? The health care provider in this patient scenario is concerned that E. O. Might have type I-hypersensitivity. The patient has indications of this reaction as written in the provided patient history. Having an allergy to bee stings, and being a diagnosed asthma patient makes the Health care provider cautious in prescribing E. O.

A course of penicillin, which is a broad spectrum antibiotic and a possible cause of human nonphysical as Indicated In the textbook 10-1 (Cooperates & Bananas, 2013).

The bee ting allergy Indicates that the patient has hypersensitivity and having asthma indicates the physiological mechanisms too type 1 reaction. Type I-hypersensitivity reactions can be systemic, which are shock like reactions and can be fatal. The reactions also can be localized, which have a specific target tissue/organ (I. E. Bronchiole constriction experienced in asthma).

If this patient has an adverse allergic reaction to the antibiotics It could result In potential harm. (DRP. Thule Vanilla’s).

A drastic case of unpractical shock because of the meds could give the patient ivies, itching, and edema prognostications and swelling of the throat, which could ultimately lead to death (Cooperates & Bananas, 2013). The health care provider cautions her parents to watch her closely for an allergic reaction to the antibiotic so that it can be reported immediately and dealt with accordingly. Explain the role of leg and mast cells In type I-hypersensitivity reactions.

Why might E_O_ react adversely to the antibiotic with the first use?

Antigen specific leg and tissue mast cell products such as histamine and prostaglandin fuel type I-hypersensitivity reactions. Genetic mechanisms influence yep I-hypersensitivity with strong hereditary linkage regarding the leg response to allergens, which are usually environmental. Mucilaginous E (leg) Is the primary antibody mediating this reaction. It Is produced by plasma cells and circulates In small amounts In the blood. It usually takes repeated exposure to cause significantly high levels of leg in the blood.

Environmental pollutants may play a role by increasing mucosa permeability and increasing antigen entry Into the body (Cooperates & Bananas, 2013). The chief effecter cell of type I-hypersensitivity is the mast cell even though they are many others. These mast cells are found in loose connective tissue and they are covered with leg receptors and are filled with vesicles or granules contacting evocative, proportionately chemical mediators that produce inflammation when released.

The leg antibody binds to the mast cell surface receptor are presented to passing antigens. The initial onset of the type I-hypersensitivity is the cross-linkage of two leg receptors to one antigen on the mast cell.

This leads to an increase in intracellular calcium that results in immediate, massive, local mast cell desegregation of preformed formalization mediators. This release causes the Inflammatory response (Cooperates & Bananas, 2013). Once the mast cell Is coated with y comes essentials to an allergic reaction.

Having a previously stated allergy to bee stings is an indication that the elaborated stated mechanism above might take place upon the penicillin consumption. The first use might cause a similar case of nonphysical and a type 1 hypersensitivity reaction.

Work Cited: Cooperates, Lee Ellen, and Jacqueline L. Bananas. Pathologically. 5th De. SST.

Louis, Mo. : Elsevier, 2013. Print. Panamanian, DRP. Third. “ Asthma & Allergy Hypersensitivity.

” N. P. , n. D. Web.

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